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This highlights the potential to exploit HO-1 as a valuable target for therapeutic intervention in DFU cases (Fig

disruptions in lipid metabolism result in lipid deposition in the renal parenchyma, promoting lipid peroxidation, further inducing ferroptosis (216)

The occurrence of fumonisin B1 was correlated with the occurrence of a higher incidence of esophageal cancer in regions of Transkei (South Africa), China, and Northeast Italy [3]